Unanticipated specificity in effector-triggered immunity

Alejandra Zárate-Potes; Hinrich Schulenburg; Katja Dierking

doi:10.1016/j.it.2024.10.008

Unanticipated specificity in effector-triggered immunity

Alejandra Zárate-Potes
, Hinrich Schulenburg^*
, Katja Dierking

^*Corresponding author for this work

Biology

Research output: Contribution to journal › Short survey › peer-review

3 Citations (Scopus)

Abstract

Effector-triggered immunity (ETI) enables hosts to react to pathogens by monitoring few key cellular processes. ETI responses are assumed to be similar toward related pathogen effectors. However, recent evidence from the invertebrate model Caenorhabditis elegans and pore-forming toxins indicates a much more complex and specific ETI than previously anticipated.

Original language	English
Pages (from-to)	939-942
Number of pages	4
Journal	Trends in Immunology
Volume	45
Issue number	12
Early online date	15 Nov 2024
DOIs	https://doi.org/10.1016/j.it.2024.10.008
Publication status	E-pub ahead of print - 15 Nov 2024

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10.1016/j.it.2024.10.008Licence: CC BY-NC

Cite this

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title = "Unanticipated specificity in effector-triggered immunity",

abstract = "Effector-triggered immunity (ETI) enables hosts to react to pathogens by monitoring few key cellular processes. ETI responses are assumed to be similar toward related pathogen effectors. However, recent evidence from the invertebrate model Caenorhabditis elegans and pore-forming toxins indicates a much more complex and specific ETI than previously anticipated.",

author = "Alejandra Z{\'a}rate-Potes and Hinrich Schulenburg and Katja Dierking",

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T1 - Unanticipated specificity in effector-triggered immunity

AU - Zárate-Potes, Alejandra

AU - Schulenburg, Hinrich

AU - Dierking, Katja

PY - 2024/11/15

Y1 - 2024/11/15

N2 - Effector-triggered immunity (ETI) enables hosts to react to pathogens by monitoring few key cellular processes. ETI responses are assumed to be similar toward related pathogen effectors. However, recent evidence from the invertebrate model Caenorhabditis elegans and pore-forming toxins indicates a much more complex and specific ETI than previously anticipated.

AB - Effector-triggered immunity (ETI) enables hosts to react to pathogens by monitoring few key cellular processes. ETI responses are assumed to be similar toward related pathogen effectors. However, recent evidence from the invertebrate model Caenorhabditis elegans and pore-forming toxins indicates a much more complex and specific ETI than previously anticipated.

UR - https://www.scopus.com/pages/publications/85209247314

UR - https://www.scopus.com/pages/publications/85209247314#tab=citedBy

U2 - 10.1016/j.it.2024.10.008

DO - 10.1016/j.it.2024.10.008

M3 - Short survey

AN - SCOPUS:85209247314

SN - 1471-4906

VL - 45

SP - 939

EP - 942

JO - Trends in Immunology

JF - Trends in Immunology

IS - 12

ER -

Unanticipated specificity in effector-triggered immunity

Abstract

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