Unanticipated specificity in effector-triggered immunity

Alejandra Zárate-Potes; Hinrich Schulenburg; Katja Dierking

doi:10.1016/j.it.2024.10.008

Unanticipated specificity in effector-triggered immunity

Alejandra Zárate-Potes, Hinrich Schulenburg^*, Katja Dierking

^*Corresponding author for this work

Biology

Research output: Contribution to journal › Short survey › peer-review

Abstract

Effector-triggered immunity (ETI) enables hosts to react to pathogens by monitoring few key cellular processes. ETI responses are assumed to be similar toward related pathogen effectors. However, recent evidence from the invertebrate model Caenorhabditis elegans and pore-forming toxins indicates a much more complex and specific ETI than previously anticipated.

Original language	English
Pages (from-to)	1-4
Journal	Trends in Immunology
Early online date	15 Nov 2024
DOIs	https://doi.org/10.1016/j.it.2024.10.008
Publication status	E-pub ahead of print - 15 Nov 2024

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10.1016/j.it.2024.10.008Licence: CC BY-NC

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title = "Unanticipated specificity in effector-triggered immunity",

abstract = "Effector-triggered immunity (ETI) enables hosts to react to pathogens by monitoring few key cellular processes. ETI responses are assumed to be similar toward related pathogen effectors. However, recent evidence from the invertebrate model Caenorhabditis elegans and pore-forming toxins indicates a much more complex and specific ETI than previously anticipated.",

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AU - Zárate-Potes, Alejandra

AU - Schulenburg, Hinrich

AU - Dierking, Katja

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N2 - Effector-triggered immunity (ETI) enables hosts to react to pathogens by monitoring few key cellular processes. ETI responses are assumed to be similar toward related pathogen effectors. However, recent evidence from the invertebrate model Caenorhabditis elegans and pore-forming toxins indicates a much more complex and specific ETI than previously anticipated.

AB - Effector-triggered immunity (ETI) enables hosts to react to pathogens by monitoring few key cellular processes. ETI responses are assumed to be similar toward related pathogen effectors. However, recent evidence from the invertebrate model Caenorhabditis elegans and pore-forming toxins indicates a much more complex and specific ETI than previously anticipated.

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Unanticipated specificity in effector-triggered immunity

Abstract

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